Scientific Program - Dementia
Please see below the CONy Scientific Program. Please click on the appropriate section (ordered by ABC) to view the relevant program. Please note that the program and timing is subject to change. To view the program timetable, please click here
|Dementia||Epilepsy||Headache||History of Neurology in Poland|
|Section Heads: Tomasz Gabryelewicz, Poland & Lefkos Middleton, UK|
|SESSION 17 | DEMENTIA: RISK FACTORS
|Chairs: Nataliya Pryanykova, Ukraine & Ricardo Nitrini, Brazil|
Does general anesthesia increase the risk of dementia?
Anecdotal evidence and limited case series report exacerbation of cognition following surgery under general anesthetics. Is this observation correct and should elderly individuals be warned against it?
|08:45-08:55||Host: Isabel Santana, Portugal|
Pro: Magda Tsolaki, Greece
|09:15-09:35||Con: Lawrence Honig, USA|
|09:35-09:50||Discussion and Rebuttals|
Is obstructive sleep apnea an important risk factor for dementia?
|Capsule:||Obstructive sleep apnea (OSA) causes brain anoxia, which theoretically can exacerbate damage to the vulnerable brain. Is this of practical importance and should patients be screened for OSA?|
|09:50-10:00||Host: Luiza Spiru, Romania|
|10:00-10:20||Pro: Giancarlo Logroscino, Italy|
|10:20-10:40||Con: Lawrence Honig, USA|
|10:40-10:55||Discussion and Rebuttals|
|11:10-13:10||SESSION 18 | ALZHEIMER'S DISEASE (AD): DIAGNOSIS AND THERAPY|
|Chair: Homa Ebrahimi, Iran & Rachelle Doody, USA|
Can the diagnosis of AD be made solely on biomarker evidence?
|Capsule:||Is the presence of CSF and/or imaging biomarker evidence of abnormal brain load of amyloid and tau sufficient for the diagnosis of AD, even in the absence of cognitive decline?|
Host: David Knopman, USA
|11:20-11:40||Pro: Giancarlo Logroscino, Italy|
|11:40-12:00||Con: Lefkos Middleton, UK|
|12:00-12:15||Discussion and Rebuttals|
Should agitation in AD be treated with antipsychotics?
Agitation in AD patients is an ominous sign, frequently leading to nursing home placement. Is it justified to treat patients with neuroleptic drugs in spite of their significant adverse effects?
Host: Tomasz Gabryelewicz, Poland
|12:25-12:45||Pro: Daniel Drubach, USA|
|12:45-13:05||Con: Pasquale Calabrese, Switzerland|
|13:05-13:20||Discussion and Rebuttals|
|13:20-14:20||Industry Sponsored Symposium (Not for CME) – HALL A|
SESSION 20| AD: CONCEPTS
|Chair: Mee Young Park, South Korea & Mirek Brys, USA|
|15:05-16:00||Is amyloid deposition a non-specific manifestation of aging?|
|Capsule:||Beta amyloid deposition is required for a pathological diagnosis of AD. Yet, many elderly people have such deposits but are cognitively normal. Can amyloid be simply a manifestation of aging, like baldness or skin wrinkles?|
Host: Lefkos Middleton, UK
|15:15-15:30||Pro: Panteleimon Giannakopoulos, Switzerland|
|15:30-15:45||Con: Marios Politis, UK|
|15:45-16:00||Discussion and Rebuttals|
|16:00-16:55||The term "Alzheimer's disease" should be dropped as it is impeding future research|
|Capsule:||For 50 years, the term AD was reserved for cases with presenile dementia, but the term has been expanded to replace the term "sentile dementia". Is this terminology shift helpful?|
Host: Robert Perneczky, UK
|16:10-16:25||Pro: Amos Korczyn, Israel|
|16:25-16:40||Con: David Knopman, USA|
|16:40-16:55||Discussion and Rebuttals|
|17:10-19:00||SESSION 21| DEMENTIA - TREATMENT|
|Chairs: Judith Aharon, Israel|
|17:10-18:05||Does cognitive stimulation have value in the treatment of mild cognitive impairment (MCI) and early dementia?|
Cognitive stimulation has been suggested as an important disease modifying strategy in people with early cognitive decline including engagement in crossword puzzles, bridge playing, and computer-based excercises. But are these really effective? What is the evidence?
Host: Dorota Religa, Sweden
Pro: Michael Geschwind, USA
|17:35-17:50||Con: Roger Bullock, UK|
|17:50-18:05||Discussion and Rebuttals|
|18:05-19:00||Is inflammation a valid target for intervention in AD?|
Inflammatory processes occur in any damaged tissue, and are seen in AD brains as activated microglia and astrocytes, as well as elevated cytokine and chemokine levels. Are these detrimental and should they be manipulated?
Host: Maria Barcikowska, Poland
Pro: Agneta Nordberg, Sweden
|18:30-18:45||Con: Panteleimon Giannakopoulos, Switzerland|
|18:45-19:00||Discussion and Rebuttals|
|SESSION 45| DEMENTIA
|Chairs: Michal Prendecki, Poland & Eugene Tarnow, USA|
Is traumatic brain injury a risk for dementia?
|Capsule:||In recent years, reports appeared associating
moderate to severe traumatic brain injury with a greater risk to develop
Alzheimer's disease or another type of dementia. Is this association true, how
strong is the risk, and what is the underlying mechanism?
|11:00-11:10||Host: Babak Tousi, USA|
Pro: Dan Perl, USA
|11:25-11:40||Con: Daniel Drubach, USA|
|11:40-12:00||Discussion and Rebuttals|
The clinical criteria of NPH are sensitive and predictive of response to shunting
Not all patients shunted for NPH can improve after surgery. Patient's clinical profile, shunt functioning, quality of surgery may contribute. Predictors for improvement are often misleading. Clinical, physiological measurement, and imaging factors can present with different pros and cons.
|12:00-12:10||Host: Marek Czonsnyka, UK|
|12:10-12:25||Pro: Ullrich Wuellner, Germany|
|12:25-12:40||Con: Carsten Wikkelso, Sweden|
|12:40-13:00||Discussion and Rebuttals|